Myocardial infarct size and cardiac performance at exercise soon after myocardial infarction.
نویسندگان
چکیده
منابع مشابه
Exercise testing soon after myocardial infarction.
Forty-six men under age 70, without clinical congestive heart failure or unstable angina pectoris, performed treadmill tests 3, 5, 7, 9 and 11 weeks after myocardial infarction. Patients were more frequently able to perform moderate exertion (2 mph, 14% grade) at 7 and 11 weeks than at 3 weeks following infarction. Ischemic ST-segment depression, usually unaccompained by angina pectoris, occurr...
متن کاملTesting Soon After Myocardial Infarction
men completed a heart-rate-limited (HRL) protocol and 107 completed a symptom-limited (SXL) protocol 3 weeks after the acute event. In the HRL protocol, effort terminated at a heart rate of 130 beats/min in the absence of a limiting symptom, exertional hypotension or ventricular tachycardia. Peak heart rate was not an end point in the SXL protocol. Despite a higher peak heart rate and work load...
متن کاملRepeated Treadmill Exercise Testing Soon After Myocardial Infarction
To determine the response to repeated treadmill exercise testing soon after uncomplicated myocardial infarction, 24 males (mean age 54 6 years) performed two symptom-limited tests several days apart 3, 7 and 11 weeks after the acute event. Significant within-week differences were noted for peak exercise tolerance (mets) and peak heart rate at 7 weeks (p < 0.05). Significant within-week differen...
متن کاملCardiac troponin T at 96 hours after acute myocardial infarction correlates with infarct size and cardiac function.
OBJECTIVES In clinical practice, myocardial infarct size can be estimated non-invasively by nuclear imaging techniques or contrast-enhanced magnetic resonance imaging (CE-MRI). Due to limited availability and high costs, serologic tests are frequently used as an alternative. BACKGROUND We examined the ability of a single value of cardiac troponin T (cTnT) 96 h after onset of ST-/non-ST-segmen...
متن کاملAnti-CCL21 antibody attenuates infarct size and improves cardiac remodeling after myocardial infarction.
BACKGROUND/AIMS Over-activation of cellular inflammatory effectors adversely affects myocardial function after acute myocardial infarction (AMI). The CC-chemokine CCL21 is, via its receptor CCR7, one of the key regulators of inflammation and immune cell recruitment, participates in various inflammatory disorders, including cardiovascular ones. This study explored the therapeutic effect of an an...
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ژورنال
عنوان ژورنال: Heart
سال: 1982
ISSN: 1355-6037
DOI: 10.1136/hrt.47.1.44